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Actavis is often a NSAID that inhibits both equally isoforms of COX enzyme, a essential enzyme involved in the inflammatory cascade. COX-one enzyme is a constitutive enzyme linked to prostacyclin generation and protective capabilities of gastric mucosa whereas COX-two is definitely an inducible enzyme linked to the production of inflammatory mediators in reaction to inflammatory stimuli. Actavis displays extra selectivity in the direction of COX-2 (IC50 of 0.77uM) than COX-one (IC50 of >100uM), which promotes its gastric tolerance as compared to other NSAIDs.

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